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E ID#: AMI-2 Performance Measure Name: Aspirin Prescribed at Discharge Description: Acute myocardial infarction (AMI) patients without aspirin contraindications who are prescribed aspirin at hospital discharge Rationale: Aspirin therapy in patients who have suffered an acute myocardial infarction reduces the risk of adverse events and mortality. Studies have demonstrated that aspirin can reduce this risk by 20% (Antiplatelet Trialists' Collaboration, 1994). National guidelines strongly recommend.
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Krumholz FAQ
Tests Electrocardiogram (ECG or EKG). This first test done to diagnose a heart attack records electrical signals as they travel through the heart. ... Blood tests. ... Chest X-ray. ... Echocardiogram. ... Coronary catheterization (angiogram). ... Cardiac computed tomography (CT) or Magnetic resonance imaging (MRI).
Cardiac biomarkers/enzymes: The American College of Cardiology/American Heart Association (ACC/AHA) and the European Society of Cardiology (ESC) guidelines recommend cardiac troponin as the only cardiac biomarker that should be measured at presentation in patients with suspected MI, due to its superior sensitivity and ...
These nine Core Measures include the administration of aspirin at arrival, the prescription of aspirin, beta-blockers and statins (as appropriate) upon discharge, the use of Angiotensin Converting Enzyme Inhibitor (ACEI) or Angiotensin Receptor Blocker (ARB) for all patients with left ventricular systolic dysfunction ( ...
The most sensitive early marker for myocardial infarction is myoglobin. Troponin levels should be measured at presentation and again 10-12 hours after the onset of symptoms. When there is uncertainty regarding the time of symptom onset, troponin should be measured at twelve hours after the presentation.
Cardiac enzymes are superior to ECG in the diagnosis of AMI. Myoglobin, FABP, and GPBB are early biomarkers in the diagnosis of AMI. TnT and TnI are late markers.
Cardiac biomarkers are helpful in the diagnosis of AMI, particularly NSTEMI. Troponin, creatine kinase-MB (CK-MB), and LDH are cardiac markers observed. The level of CK-MB, an isoenzyme of creatine kinase usually found in the myocardium, reaches its peak at 10 hours and normalizes within 2 to 3 days.
The gold standard for measuring infarct size is to directly quantify the amount of scar in a pathology specimen. Animal models of permanent occlusion13 and reperfusion14 have shown that the 99mTc sestamibi defect size accurately measures infarct size.
Early treatment within 6 hours of symptom onset significantly improves prognosis. An MI is diagnosed when 2 of the following criteria are met: Symptoms of ischemia. New ST-segment changes or a left bundle branch block (LBBB)
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