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The Long Form Of Fas Apoptotic Inhibitory Molecule Is Expressed ... - Jneurosci
Get The Long Form Of Fas Apoptotic Inhibitory Molecule Is Expressed ... - Jneurosci
Rons and Protects Them against Death Receptor-Triggered Apoptosis Miguel F. Segura,1 Carme Sole,1 Marta Pascual,3 Rana S. Moubarak,2 M. Jose Perez-Garcia,1 Raffaella Gozzelino,1 Victoria Iglesias,2 Nahuai Badiola,2 Jose R. Bayascas,1 Nuria Llecha,1 Jose Rodriguez-Alvarez,2 Eduardo Soriano,3 Victor J. Yuste,1 and Joan X. Comella1,2 1Cell Signaling and Apoptosis Group, Departament de Cie ncies Me diques Ba siques, Universitat de Lleida-Hospital Universitari Arnau de Vilanova, 25198 Lleida, Spai.
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Transfected FAQ
The Fas receptor is a death receptor on the surface of cells that leads to programmed cell death (apoptosis) if it binds its ligand, Fas ligand (FasL). It is one of two apoptosis pathways, the other being the mitochondrial pathway.
Fas can trimerize and transduce proapoptotic signals upon binding of its ligand, FasL. Similar to TNF, the physiological ligand for Fas (CD95L or FasL) is synthesized as a type II membrane protein and is expressed on activated B cells, T cells, and NK cells.
FAS (Fas Cell Surface Death Receptor) is a Protein Coding gene. Diseases associated with FAS include Autoimmune Lymphoproliferative Syndrome and Lymphoproliferative Syndrome. Among its related pathways are CASP8 activity is inhibited and TNFR1 Pathway.
Fas ligand or FasL is a homotrimeric type II transmembrane protein that belongs to the tumor necrosis factor (TNF) family. It signals through trimerization of FasR, which spans the membrane of the target cell. This trimerization usually leads to apoptosis, or programmed cell death.
Fas ligand (FasL) is a homotrimeric membrane molecule on the regulator cells that bind three Fas molecules on target cells. Clustering of Fas receptor (FasR) molecules brings intracellular death domains and adaptor proteins close to create a death-inducing signal complex (DISC).
Fas was first identified using a monoclonal antibody generated by immunizing mice with the FS-7 cell line. Thus, the name Fas is derived from FS-7-associated surface antigen.
FAS (also known as APO-1 or CD95) belongs to the subgroup of the tumor necrosis factor receptor (TNF-R) family that contain an intra-cellular 'death domain' and can trigger apoptosis. Its physiological ligand, FASL (CD95L), is a member of the corresponding TNF cytokine family.
Fas ligand (FasL/CD95L)-mediated 'activation-induced cell death' (AICD) of T cells helps to downsize normal immune responses. AICD can occur by 'suicide' (where FasL triggers Fas (CD95) on the same T cell) or by 'fratricide' (where FasL on a T cell triggers Fas on a neighbouring T cell).
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